PACE-PACING AND CLINICAL ELECTROPHYSIOLOGY, cilt.36, sa.10, ss.1220-1227, 2013 (SCI-Expanded)
BackgroundSystemic inflammation is accepted as one of the pathophysiological mechanisms of atrial fibrillation (AF). The role of inflammation has been shown previously. Interleukin (IL) system is the main modulator of the inflammatory responses and genetic polymorphisms of IL-1 cluster genes are associated with increased risk for inflammatory diseases.