JOURNAL OF SURGICAL RESEARCH, cilt.126, sa.1, ss.73-81, 2005 (SCI-Expanded)
Background. Sepsis, commonly associated with enhanced generation of reactive oxygen metabolites, leads to multiple organ dysfunctions. The neurohypophyseal hormone oxytocin (OT), released during social contact, was recently shown to modulate the immune and inflammatory processes. We investigated the protective role of OT against sepsis-induced pelvic inflammation.